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case western reserve university

ATIT LAB

Department of Biology

 
 

PUBLICATIONS

Preethi Mani, Andrew Jarrell, John Myers, Radhika Atit (2009). Visualizing canonical Wnt signaling during mouse craniofacial development. Developmental Dynamics, Aug. 28, epub.

Knothe Tate, ML; Falls, TD; McBride, SH; Atit, R., Knothe, UR (2008). Mechanical modulation of osteochondroprogenitor cell fate. Int. J. Biochem Cell Biol. 40 (12): 27020-38. (Invited Review).

Jennifer Ohtola, John Myers, Batool Akhtar-Zaidi, Diana Zuzindlak, Pooja Sandesara, Karen Yeh, Susan Mackem, and Radhika Atit (2008). Beta-Catenin has sequential roles in the survival and specification of ventral dermis. Development 135:2321-2329.

Liu F, Chu EY, Watt B, Zhang Y, Gallant NM, Andl T, Yang SH, Lu MM, Piccolo S, Schmidt-Ullrich R, Taketo MM, Morrisey EE, Atit R, Dlugosz AA, Millar SE. (2008). Wnt/beta-catenin signaling directs multiple stages of tooth morphogenesis. Developmental Biology  313 p210-24

Atit R., Sgaier, S. Mohammed, O., Taketo, M., Dufort, D., Joyner, A., Niswander, L., Conlon, R. (2006). Beta-catenin activity is necessary and sufficient for dorsal dermal fate specification. in mouse. Developmental Biology, 296, p164-176.  

Atit, R. , Conlon, R, and Niswander, L. (2003).  EGFR signaling patterns the feather array by promoting interbud fate. Developmental Cell  4  p231-40


Rizvi, T.A, Ling, B., Huang, Y., Sidani, A., Atit, R., Largaespada, D.A., Boissy, R.E., and Ratner, N. (2002). A novel cytokine pathway suppresses glial cell melanogenesis after injury to adult nerve. J. Neuroscience  22 p.9831-40


Sherman, LS., Atit, R., Rosenbaum, T., Cox, A.D., Ratner, N. (2000). Single cell Ras-GTP analysis reveals altered Ras activity in a subpopulation of neurofibroma Schwann cells but not fibroblasts. J Biol Chem., 275 p.30740-5.


Atit, R., Mitchell, K., Nguyen, L., Warshawksy, D., Ratner, N. (2000). The neurofibromatosis type 1 (Nf1) tumor suppressor is a modifier of carcinogen-induced pigmentation and papilloma formation in C57Bl/6 mice. Accepted at J. Invest. Dermatology, 114 p.1093-1100.


Atit, R., Crowe, M., Wenstrup, R., Greenhalgh, D., and Ratner, N. (1999). The Nf1, tumor suppressor gene is required for normal skin wound healing, fibroblast proliferation, and collagen deposition. J. Invest. Dermatology, 112 p.835-842.